Example: 30 and 42 Factor them. 2 x 3 x 5 = 30 2 x 3 x 7 = 42 Combine the factors, eliminating duplicates. 2 x 3 x 5 x 7 = 210, the LCM The LCD is the same process, except it's a denominator.
(45)2 = (45) x (45) = 2,025Because that's the definition of the 'square' process.
You must use the process of "multiplication"that you learned in the 5th or 6th grade.
For example: x2-49 = (x-7)(x+7) For example: 16x2-25 = (4x-5)(4x+5)
You could use the process of multiplication. It's just a suggestion.
this is the process by which one of the two copies of the X-chromosomes present in female animals is inactivated
An X-inactivation occurs when one X-chromosome is randomly turned off and forms a Barr body. It is also known as lyonization.
its not inactive
X chromosome inactivation
Yes, testing the mother for skewed x-inactivation.
i really dont know but i have the same question and was hoping you could answer it
X chromosome inactivation happens in female mammals.
Male carry XY genotype whereas female has XX. Two copies of X chromosome means, there is a great chance of potentially toxic double dose of X-linked genes. It is known that 1000s of genes responsible for growth and cell viability are there in X chromosomes.To avoid the over production of these genes in female (where 2 X chromosomes present), there is a mechanism evolved that transcriptionally control or silence on of these two X chromosome.http://www.nature.com/scitable/topicpage/x-chromosome-x-inactivation-323
The difference between them is that : In genomic imprinting : one of the alleles in the two chromosomes ( for example the color of the eyes ) are tend to be heavily methylated, in contrast to the nonimpringting copy of the allele which typically is not methylated. In X-chromosome in activation : As you know in female there are two X chromosomes, so one of the two X xhromosomes is methylated which mean one of the two chromosomes become inactivation.
aggregation and inactivation are some of the major problems hindering the prop[er production and expression of proteins. insulin can be artificially cheperoned to reduce aggregation and inactivation.- leninkandasamy@gmail.com
X-linked genes are found on the sex X chromosome. X-linked genes just like autosomal genes have both dominant and recessive types. Recessive X-linked disorders are rarely seen in females and usually only affect males. This is because males inherit their X chromosome and all X-linked genes will be inherited from the maternal side. Fathers only pass on their Y chromosome to their sons, so no X-linked traits will be inherited from father to son. Females express X-linked disorders when they are homozygous for the disorder and become carriers when they are heterozygous. An infamous recessive X-linked disorder is Hemophilia A. Hemophilia is a disorder where blood does not clot properly due to a shortage of clotting factor VIII. This disorder gained recognition as it traveled through royal families, notably the descendent's of Britain's Queen Victoria. X-linked dominant inheritance will show the same phenotype as a heterozygote and homozygote. Just like X-linked inheritance, there will be a lack of male-to-male inheritance, which makes it distinguishable from autosomal traits. One example of a X-linked trait is Coffin-Lowry syndrome, which is caused by a mutation in ribosomal protein gene. This mutation results in skeletal, craniofacial abnormalities, mental retardation, and short stature. X chromosomes in females undergo a process known as X inactivation. X inactivation is when one of the two X chromosomes in females is almost completely inactivated. It is important that this process occurs otherwise a woman would produce twice the amount of normal X chromosome proteins. The mechanism for X inactivation will occur during the embryonic stage. For people with disorders like trisomy X, where the genotype has three X chromosomes, X-inactivation will inactivate all X chromosomes until there is only one X chromosome active. X inactivation is not only limited to females, males with Klinefelter syndrome, who have an extra X chromosome, will also undergo X inactivation to have only one completely active X chromosome.
Since one X chromosome in each cell is randomly inactivated at some point during development, a woman heterozygous for hypertrichosis would have varying regions of patchy hair if the alleles responsible for the trait were located on the X chromosome. Variation would look similar to the varied tortoise shell coat color pattern on female cats, where the presence of yellow and black (red and gold/blue and cream) is controlled by X chromosome inactivation.