Hypokalemic periodic paralysis is characterized by low levels of potassium in the blood during attacks, leading to muscle weakness or paralysis. In contrast, hyperkalemic periodic paralysis is characterized by high levels of potassium during episodes, resulting in muscle stiffness or weakness. Both conditions are genetic and involve dysfunction in ion channels that regulate potassium levels in muscle cells.
Another name for hyperkalemic periodic paralysis is Hyperkalemic Periodic Paralysis.
The gene for hyperkalemic PP affects virtually all who inherit it, with no difference in male-vs.-female expression.
Hyperkalemic periodic paralysis flare-ups typically last from a few hours to a few days. The duration can vary from person to person and can be influenced by factors such as the trigger of the flare-up and the individual's overall health. It is important for individuals experiencing flare-ups to work closely with their healthcare provider to manage symptoms and prevent future episodes.
The normal gene is responsible for a muscle protein controlling the flow of sodium during muscle contraction.
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There are two major forms, hypokalemic and hyperkalemic, each caused by defects in different genes.
Periodic paralysis disorders are genetic disorders that affect muscle strength. There are two major forms, hypokalemic and hyperkalemic, each caused by defects in different genes.
Another name for hyperkalemic periodic paralysis is Hyperkalemic Periodic Paralysis.
The gene for hyperkalemic PP affects virtually all who inherit it, with no difference in male-vs.-female expression.
The gene for hyperkalemic PP affects virtually all who inherit it, with no difference in male-vs.-female expression.
With hypokalemic periodic paralysis an attack of weakness can be induced by administering glucose and insulin with exercise. These tests are potentially hazardous and require careful monitoring.
The level of potassium in the blood rises slightly or is normal.
The level of potassium in the blood falls in the early stages of a paralytic attack.
An attack of hyperkalemic PP can be induced with administration of potassium after exercise during fasting. These tests are potentially hazardous and require careful monitoring.
The gene for hypokalemic PP is present equally in both sexes, but leads to noticeable symptoms more often in men than in women.
The gene for hypokalemic PP is present equally in both sexes, but leads to noticeable symptoms more often in men than in women.
Attacks requiring treatment are much less common in hyperkalemic PP. Glucose and insulin may be prescribed. Eating carbohydrates may also relieve attacks.